A cell cycle-associated change in Ca²⁺ releasing activity leads to the generation of Ca²⁺ transients in mouse embryos during the first mitotic division
Calmodulin-dependent protein kinase II, and not protein kinase C, is sufficient for triggering cell-cycle resumption in mammalian eggs
Different forms of glycine- and GABAᴀ-receptor mediated inhibitory synaptic transmission in mouse superficial and deep dorsal horn neurons
Distinct physiological mechanisms underlie altered glycinergic synaptic transmission in the murine mutants spastic, spasmodic, and oscillator
Drug delivery to the human and mouse uterus using immunoliposomes targeted to the oxytocin receptor
Functional maturation of mouse epididymal spermatozoa
- Jensen, L. R., Amende, M., Gurok, U., Moser, B., Gimmel, V., Tzschach, A., Janecke, A. R., Tariverdian, G., Chelly, J., Fryns, J. P., Van Esch, H., Kleefstra, T., Hamel, B., Moraine, C., Gecz, J., Turner, Gillian, Reinhardt, R., Kalscheuer, V. M., Ropers, H. H., Lenzner, S.
Premature dyad separation in meiosis II is the major segregation error with maternal age in mouse oocytes
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