Human melanoma cells selected for resistance to apoptosis by prolonged exposure to tumor necrosis factor-related apoptosis-inducing ligand are more vulnerable to necrotic cell death induced by cisplatin
Adaptation to ER stress as a driver of malignancy and resistance to therapy in human melanoma
Up-regulation of Mcl-1 is critical for survival of human melanoma cells upon endoplasmic reticulum stress
2-Deoxy-D-glucose enhances TRAIL-induced apoptosis in human melanoma cells through XBP-1-mediated up-regulation of TRAIL-R2
Metabolic approaches to treatment of melanoma
Treatment combinations targeting apoptosis to improve immunotherapy of melanoma
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