Stromal liver kinase B1 STK11 signaling loss induces oviductal adenomas and endometrial cancer by activating mammalian target of Rapamycin Complex 1
Age related increase in mTOR activity contributes to the pathological changes in ovarian surface epithelium
Germ cell specific overactivation of WNT/βcatenin signalling has no effect on folliculogenesis but causes fertility defects due to abnormal foetal development
Germ cell-specific sustained activation of Wnt signalling perturbs spermatogenesis in aged mice, possibly through non-coding RNAs
Ovarian hormones through Wnt signalling regulate the growth of human and mouse ovarian cancer initiating lesions
In vivo genetic cell lineage tracing reveals that oviductal secretory cells self-renew and give rise to ciliated cells
Inhibition of extracellular matrix mediated TGF-β signalling suppresses endometrial cancer metastasis
Overactive mTOR signaling leads to endometrial hyperplasia in aged women and mice
- Jamaluddin, M. Fairuz B., Ko, Yi-An, Scott, Rodney J., Nahar, Pravin, Tanwar, Pradeep S., Kumar, Manish, Brown, Yazmin, Bajwa, Preety, Nagendra, Prathima B., Skerrett-Byrne, David A., Hondermarck, Hubert, Baker, Mark A., Dun, Matthew D.
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