Exploring how low oxygen post conditioning improves stroke-induced cognitive impairment: a consideration of amyloid-beta loading and other mechanisms
- Zhao, Zidan, Hood, Rebecca, Ong, Lin Kooi, Pietrogrande, Giovanni, Sanchez Bezanilla, Sonia, Warren, Kirby E., Ilicic, Marina, Kluge, Murielle G., TeBay, Clifford, Ottersen, Ole P., Johnson, Sarah J., Nilsson, Michael, Walker, Frederick R.
Low oxygen post conditioning prevents thalamic secondary neuronal loss caused by excitotoxicity after cortical stroke
Involvement of microglia activation in the development of CNS diseases
Growth hormone improves cognitive function after experimental stroke
- Ong, Lin Kooi, Chow, Wei Zhen, Walker, Frederick R., Nilsson, Michael, Isgaard, Jörgen, Tebay, Clifford, Kluge, Murielle, Pietrogrande, Giovanni, Zalewska, Katarzyna, Crock, Patricia, Åberg, Daivd, Bivard, Andrew, Johnson, Sarah J.
Sustained administration of corticosterone at stress-like levels after stroke suppressed glial reactivity at sites of thalamic secondary neurodegeneration
Corticosterone administration alters white matter tract structure and reduces gliosis in the sub-acute phase of experimental stroke
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