Chronic stress exposure following photothrombotic stroke is associated with increased levels of Amyloid beta accumulation and altered oligomerisation at sites of thalamic secondary neurodegeneration in mice
Chronic stress induced disruption of the peri-infarct neurovascular unit following experimentally induced photothrombotic stroke
Early life peripheral lipopolysaccharide challenge reprograms catecholaminergic neurons
Functional programming of the autonomic nervous system by early life immune exposure: implications for anxiety
Growth hormone deficiency is frequent after recent stroke
Growth hormone improves cognitive function after experimental stroke
- Ong, Lin Kooi, Chow, Wei Zhen, Walker, Frederick R., Nilsson, Michael, Isgaard, Jörgen, Tebay, Clifford, Kluge, Murielle, Pietrogrande, Giovanni, Zalewska, Katarzyna, Crock, Patricia, Åberg, Daivd, Bivard, Andrew, Johnson, Sarah J.
Is stroke a neurodegenerative condition? a critical review of secondary neurodegeneration and amyloid-beta accumulation after stroke
Neurobiological consequences of stress: tyrosine hydroxylase phosphorylation in response to stress
Peripheral lipopolysaccharide challenge induces long-term changes in tyrosine hydroxylase regulation in the adrenal medulla
Reconsidering the role of glial cells in chronic stress-induced dopaminergic neurons loss within the substantia nigra? Friend or foe?
Sustained administration of corticosterone at stress-like levels after stroke suppressed glial reactivity at sites of thalamic secondary neurodegeneration
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