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Translational control of tumor necrosis factor-related apoptosis-inducing ligand death receptor expression in melanoma cells

Human melanoma cells selected for resistance to apoptosis by prolonged exposure to tumor necrosis factor-related apoptosis-inducing ligand are more vulnerable to necrotic cell death induced by cisplatin

Progression in melanoma is associated with decreased expression of death receptors for tumor necrosis factor–related apoptosis-inducing ligand

Activation of Jun N-terminal kinase is a mediator of vincristine-induced apoptosis of melanoma cells

Adaptation to ER stress as a driver of malignancy and resistance to therapy in human melanoma

Inhibition of apoptosis facilitates necrosis induced by cisplatin in gastric cancer cells

Inhibition of endoplasmic reticulum stress-induced apoptosis of melanoma cells by the ARC protein

Involvement of endoplasmic reticulum stress in docetaxel-induced JNK-dependent apoptosis of human melanoma

Melanoma cell sensitivity to docetaxal-induced apoptosis is determined by class III β-tubulin levels

Overcoming resistance to apoptosis in cancer therapy

Small molecular weight variants of p53 are expressed in human melanoma cells and are induced by the DNA-damaging agent cisplatin

Up-regulation of Mcl-1 is critical for survival of human melanoma cells upon endoplasmic reticulum stress

2-Deoxy-D-glucose enhances TRAIL-induced apoptosis in human melanoma cells through XBP-1-mediated up-regulation of TRAIL-R2

Expression of glucose-regulated stress protein GRP78 is related to progression of melanoma

Glucose-regulated protein 78 antagonizes cisplatin and adriamycin in human melanoma cells

Human melanoma cells under endoplasmic reticulum stress acquire resistance to microtubule-targeting drugs through XBP-1-mediated activation of Akt

Human melanoma cells under endoplasmic reticulum stress are more susceptible to apoptosis induced by the BH3 mimetic obatoclax

Inhibition of MEK blocks GRP78 up-regulation and enhances apoptosis induced by ER stress in gastric cancer cells

Metabolic approaches to treatment of melanoma

Treatment combinations targeting apoptosis to improve immunotherapy of melanoma

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