A cell cycle-associated change in Ca²⁺ releasing activity leads to the generation of Ca²⁺ transients in mouse embryos during the first mitotic division
A cytosolic sperm protein factor mobilizes Ca²⁺ from intracellular stores by activating multiple Ca²⁺ release mechanisms independently of low molecular weight messengers
A novel mechanism controls the Ca²⁺ oscillations triggered by activation of ascidian eggs and has an absolute requirement for Cdk1 activity
APC FZR1 prevents nondisjunction in mouse oocytes by controlling meiotic spindle assembly timing
Application of two-photon flash photolysis to reveal intercellular communication and intracellular Ca²⁺ movements
Calmodulin-dependent protein kinase II, and not protein kinase C, is sufficient for triggering cell-cycle resumption in mammalian eggs
Cell cycle-dependent repetitive Ca²⁺ waves induced by a cytosolic sperm extract in mature ascidian eggs mimic those observed at fertilization
Are you sure you would like to clear your session, including search history and login status?